Please use this identifier to cite or link to this item:
http://hdl.handle.net/10397/106193
DC Field | Value | Language |
---|---|---|
dc.contributor | Department of Applied Biology and Chemical Technology | en_US |
dc.creator | Chen, JW | en_US |
dc.creator | Chen, SB | en_US |
dc.creator | Chen, GQ | en_US |
dc.date.accessioned | 2024-05-03T00:45:43Z | - |
dc.date.available | 2024-05-03T00:45:43Z | - |
dc.identifier.uri | http://hdl.handle.net/10397/106193 | - |
dc.language.iso | en | en_US |
dc.publisher | OAE Publishing Inc | en_US |
dc.rights | © The Author(s) 2023. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, sharing, adaptation, distribution and reproduction in any medium or format, for any purpose, even commercially, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. | en_US |
dc.rights | The following publication Chen JW, Chen S, Chen GQ. Recent advances in natural compounds inducing non-apoptotic cell death for anticancer drug resistance. Cancer Drug Resist 2023;6:729-47 is available at https://dx.doi.org/10.20517/cdr.2023.78. | en_US |
dc.subject | Drug resistance | en_US |
dc.subject | Cancer therapy | en_US |
dc.subject | Natural compound | en_US |
dc.subject | Non-apoptotic cell death | en_US |
dc.title | Recent advances in natural compounds inducing non-apoptotic cell death for anticancer drug resistance | en_US |
dc.type | Journal/Magazine Article | en_US |
dc.identifier.spage | 729 | en_US |
dc.identifier.epage | 747 | en_US |
dc.identifier.volume | 6 | en_US |
dc.identifier.doi | 10.20517/cdr.2023.78 | en_US |
dcterms.abstract | The induction of cell death is recognized as a potent strategy for cancer treatment. Apoptosis is an extensively studied form of cell death, and multiple anticancer drugs exert their therapeutic effects by inducing it. Nonetheless, apoptosis evasion is a hallmark of cancer, rendering cancer cells resistant to chemotherapy drugs. Consequently, there is a growing interest in exploring novel non-apoptotic forms of cell death, such as ferroptosis, necroptosis, pyroptosis, and paraptosis. Natural compounds with anticancer properties have garnered significant attention due to their advantages, including a reduced risk of drug resistance. Over the past two decades, numerous natural compounds have been discovered to exert anticancer and anti-resistance effects by triggering these four non-apoptotic cell death mechanisms. This review primarily focuses on these four non-apoptotic cell death mechanisms and their recent advancements in overcoming drug resistance in cancer treatment. Meanwhile, it highlights the role of natural compounds in effectively addressing cancer drug resistance through the induction of these forms of non-apoptotic cell death. | en_US |
dcterms.accessRights | open access | en_US |
dcterms.bibliographicCitation | Cancer drug resistance, 2023, v. 6, p. 729-747 | en_US |
dcterms.isPartOf | Cancer drug resistance | en_US |
dcterms.issued | 2023 | - |
dc.identifier.isi | WOS:001090499400003 | - |
dc.identifier.eissn | 2578-532X | en_US |
dc.description.validate | 202405 bcrc | en_US |
dc.description.oa | Version of Record | en_US |
dc.identifier.FolderNumber | OA_Scopus/WOS | - |
dc.description.fundingSource | Others | en_US |
dc.description.fundingText | Shenzhen Science and Technology Innovation Commission | en_US |
dc.description.fundingText | Innovation and Technology Fund -Mainland -Hong Kong Joint Funding Scheme | en_US |
dc.description.fundingText | Research Centre for Chinese Medicine Innovation of The Hong Kong Polytechnic University | en_US |
dc.description.fundingText | Hong Kong Polytechnic University Start-up Fund | en_US |
dc.description.pubStatus | Published | en_US |
dc.description.oaCategory | CC | en_US |
Appears in Collections: | Journal/Magazine Article |
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File | Description | Size | Format | |
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cdr6078_down.pdf | 2.98 MB | Adobe PDF | View/Open |
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