Please use this identifier to cite or link to this item:
http://hdl.handle.net/10397/62109
DC Field | Value | Language |
---|---|---|
dc.contributor | School of Nursing | - |
dc.creator | Yu, SS | - |
dc.creator | Tang, X | - |
dc.creator | Ho, YS | - |
dc.creator | Chang, RCC | - |
dc.creator | Chiu, K | - |
dc.date.accessioned | 2016-12-19T08:58:37Z | - |
dc.date.available | 2016-12-19T08:58:37Z | - |
dc.identifier.uri | http://hdl.handle.net/10397/62109 | - |
dc.language.iso | en | en_US |
dc.publisher | Frontiers Research Foundation | en_US |
dc.rights | Copyright: © 2016 Yu, Tang, Ho, Chang and Chiu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY) (https://creativecommons.org/licenses/by/4.0/). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms. | en_US |
dc.rights | The following publication Yu SS, Tang X, Ho Y-S, Chang RC-C and Chiu K (2016) Links between the Brain and Retina: The Effects of Cigarette Smoking-Induced Age-Related Changes in Alzheimer’s Disease and Macular Degeneration. Front. Neurol. 7:119,1-4 is available at https://dx.doi.org/10.3389/fneur.2016.00119 | en_US |
dc.subject | Age-related macular degeneration | en_US |
dc.subject | Alzheimer-like pathology | en_US |
dc.subject | Amyloid peptide | en_US |
dc.subject | Cigarette smoking | en_US |
dc.subject | Retina | en_US |
dc.title | Links between the brain and retina : the effects of cigarette smoking-induced age-related changes in Alzheimer's disease and macular degeneration | en_US |
dc.type | Journal/Magazine Article | en_US |
dc.identifier.spage | 1 | en_US |
dc.identifier.epage | 4 | en_US |
dc.identifier.volume | 7 | en_US |
dc.identifier.doi | 10.3389/fneur.2016.00119 | en_US |
dcterms.abstract | Alzheimer's disease (AD) is characterized by the progressive and selective loss of neurons and synapses. This debilitating disease was estimated to affect 33.9 million patients worldwide in 2011, a number that is expected to triple over the next 40 years (1). It has been shown that a combination of several processes, including extracellular deposition of amyloid-beta (Aß) plaques and the formation of intracellular neurofibrillary tangles (NFTs) composed of hyper-phosphorylated tau proteins, in the brain are involved in the declining cognitive processes associated with AD (2). While dysfunction of the aforementioned biological processes starts from Braak stage I (preclinical AD), it is not until the majority of the neocortex is severely affected by neurofibrillary changes (Braak stages V-VI) that patients are ultimately diagnosed with dementia (3). | - |
dcterms.accessRights | open access | en_US |
dcterms.bibliographicCitation | Frontiers in neurology, 27 July 2016, v. 7, 119, p. 1-4 | - |
dcterms.isPartOf | Frontiers in neurology | - |
dcterms.issued | 2016-07-27 | - |
dc.identifier.isi | WOS:000380341300001 | - |
dc.identifier.scopus | 2-s2.0-84982225578 | - |
dc.identifier.pmid | 27512384 | - |
dc.identifier.eissn | 1664-2295 | en_US |
dc.identifier.artn | 119 | en_US |
dc.description.oa | Version of Record | en_US |
dc.identifier.FolderNumber | OA_IR/PIRA | en_US |
dc.description.pubStatus | Published | en_US |
Appears in Collections: | Journal/Magazine Article |
Files in This Item:
File | Description | Size | Format | |
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Yu_Brain_Retina_Cigarette.pdf | 122.78 kB | Adobe PDF | View/Open |
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